MediaWiki API result

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            "588": {
                "pageid": 588,
                "ns": 0,
                "title": "Re-infection of the mosquito",
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                        "*": "----\n'''Navigation'''</br>\n<span style=\"font-size:80%\">(click blue highlighted text to return to page)</span></br></br>\n<span style=\"font-size:90%\">[[Malaria Index|Malaria main index]]</span></br>\n<span style=\"font-size:90%\">>[[Malaria Biology|Basic malaria biology]]</span></br>\n<span style=\"font-size:90%\">>>This page: <u>''Re-infection of the mosquito</u></span>\n----\n\n\n{| class=\"wikitable\" style=\"border-style: solid; border-width: 5px; border-color: #023020; color:black\"\n|colspan=\"1\" style = \"font-size:100%; color:black; background: #afbddb |'''Gametogenesis and sexual replication'''\n|}\n\n<gallery mode=\"nolines\" widths=300px heights=300px>\nFile:Mosquito uptake.jpg|<span style=\"font-size:90%\">''Development within the the mosquito''</span>|link={{filepath:Mosquito uptake.jpg}}\n</gallery>\n\nWhen a mosquito feeds from a malaria infected host, the blood meal will contain parasites of all forms. Importantly these forms include male and female gametocytes. The pH and temperature changes with the mosquito midgut cause the gametocytes to gain full sexual maturity (gametogenesis). The male gametocyte then undergoes 3 cycles of division to generate 8 male sexual forms (gametes). These then 8 gametes then \"break out\" of the male microgametocytes in a process known as exflagellation, and fuse with the female oocyte (derived from the macrogametocyte). Then following successful fusion move to the next stage of development with the mosquito gut wall. Finally the parasite enter the salivary glands of the mosquito as sporozoites where a new infection can be initiated.  \n\n----\n\n\n{| class=\"wikitable\" style=\"border-style: solid; border-width: 5px; border-color: #023020; color:black\"\n|colspan=\"1\" style = \"font-size:100%; color:black; background: #afbddb |'''Relevance to blood'''\n|}\n\nIf samples of blood undergo delayed spreading then the changes to pH and temperature caused by storage may partly mimic the conditions within the mosquito midgut leading to changes that would only normally be seen in the mosquito. Some may be difficult to recognise but are clearly atypical in appearance, others such as \"exflagellation\" have very clear appearances. It is important to be able to recognise these forms as they can cause confusion and mis-diagnosis.\n\n\n\n<gallery mode=\"nolines\" widths=240px heights=240px>\nFile:ExFl4.jpg|<span style=\"font-size:80%\"></span>|link={{filepath:ExFl4.jpg}}\n</gallery>\n<span style=\"font-size:90%\">''The gametes of ''P.falciparum'' are seen in the process of exflagellation from  the male gametocyte in a sample that has undergone delayed processing''</span>\n\n\n<span style=\"font-size:200%\">&#x2192;</span>[[Images of gametocyte development in stored blood|Click for additional clinical images]]\n\n\n----"
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            "398": {
                "pageid": 398,
                "ns": 0,
                "title": "Recognition of APL differentiation",
                "revisions": [
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                        "*": "----\n<div style=\"width: 200px\">\n{| class=\"wikitable\" style=\"border-left:solid 5px green;border-right:solid 5px green;border-top:solid 5px black;border-bottom:solid 5px black; font-size:90%; color:navy\"\n| colspan=\"1\"''|[[Atypical patterns of primitive marker expression in acute myeloid leukaemia|Return to previous page]]''\n|}\n</div>\n\n----\n\n\n<span style=\"font-size:90%\">'''Recognition of acute promyelocytic leukaemia'''</span>\n\n[[Image:AML M1.png|130px]]  \n\n<span style=\"font-size:90%\">Typical APL has cells that are hypergranular (A) or may be the microgranular variant (B) generally the cells will have a range of granulation or nuclear appearance. While CD45 expression tends to be weak, in almost all cases the variable morphological features cause a wide range of side-scatter with the abnormal cells often forming a \"cloud\" extending toward the neutrophil area. This can make the cells (particular hypergranular forms) difficult to distinguish from reactive myeloid populations (or occasionally from MDS). Absent HLA-DR on APL cells can help distinguish from reactive cells (expressing HLA-DR), but correlation with morphology and clinical features and other confirmatory tests are essential to the recognition of APL.''</span>\n\n----"
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